...that they're talking about in utero development.

All embryos, XX and XY, have the potential to make a uterus and fallopian tubes; the "gonads" can develop into either ovaries or testicles. It all depends on being exposed to the right signals and chemicals during fetal development. Once you're past that critical time, it can't be done. Kind of like once your hip bones are set, no amount of estrogen will make the pelvis "womanly", except for fat deposition around it. But expose a child to estrogen between age 8 - 14, and it's possible. So, for right now, no "protein shot" will be able to give us female fertility.

Now cloning, however... since cloning makes organs as if they were being developed in utero, that holds some promise. Imagine them making you a uterus and tubes and even ovaries from your own stem cells. Not a reality - but maybe not that far off?

I apologize if some of the language above is science lingo. I generally try to eschew obfuscation - **Sigh**

The first article indicated that disabling the Dmrt1 gene in mice caused many (but not all) of the testis cells to take on a role more like those of ovarian cells. In terms of what could feasibly happen if the same process applied in humans (animal tests don't always produce the same effects when applied to humans!) and all were converted, self-production of estrogen and progesterone (and possibly the cyclical nature of them) may be the full extent: in GGs oocytes are formed during embryonic development (could a primordial oocyte be triggered to undergo maturation when it was a few months old rather than 12+ years?); while of course scrotal ovaries wouldn't be in the same location or have surrounding structures (e.g. fallopian tubes, uterus) necessary for the menstrual cycle.

As for the second article, sadly that's completely irrelevant - Gadd45g and Sry are primarily expressed during embryonic development, so disabling them after they'd been expressed would probably have negligible effect.