The Chinese CDC published extensive data about their experience with the outbreak. Men, indeed, were dying at a ratio of 2.7:1 (which according to my calculator is 73%). However, smokers were dying at a much higher rate than nonsmokers, and men in that province smoke at a much higher rate than women, which might account for the difference.

At the risk of spoiling the double-entendre, obviously the diseases that are "other" are other than CoVID-19, not the Y-chromosome.

Shifting further into serious discussion, there's been quite a lot of discussion in medical circles around the poor survival of people with high blood pressure. A couple of theories have to do with a cell receptor (ACE2) which is the entry target for the virus, as well as directly involved in hypertension. Two classes of drugs target that receptor to control hypertension. One is a receptor blocker. The other inhibits the enzyme that normally targets the receptor. Over time, use of either of them causes the body to compensate by making more receptors. To make things more complicated, that receptor is also involved in the immune system response to infection, and what's killing most of the patients is some kind of wild over-reaction that the body can't shut off before the lungs drown.

Diagnosed with hypertension for over 50 years, I'm a prime example of someone who could be in big trouble if I contract this virus. My doctor just switched me from the inhibitor to the blocker on a hunch, but nobody knows if it's a good idea in real life.